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  1. 1
    "The sequence of the human genome."
    Venter J.C., Adams M.D., Myers E.W., Li P.W., Mural R.J., Sutton G.G., Smith H.O., Yandell M., Evans C.A., Holt R.A., Gocayne J.D., Amanatides P., Ballew R.M., Huson D.H., Wortman J.R., Zhang Q., Kodira C.D., Zheng X.H.
    Zhu X.
    Science 291:1304-1351(2001) [PubMed] [Europe PMC] [Abstract]
    Category: Sequences.
    Source: UniProtKB/TrEMBL (unreviewed).

    This publication is cited by 8214 other entries.

  2. 2
    "The nucleotide sequence of a long cDNA clone isolated from human spleen."
    Jikuya H., Takano J., Nomura N., Kikuno R., Nagase T., Ohara O.
    Submitted (JAN-2002) to the EMBL/GenBank/DDBJ databases
    Category: Sequences.
    Tissue: Spleen.
    Source: UniProtKB/TrEMBL (unreviewed).
  3. 3
    Category: Sequences.
    Source: UniProtKB/TrEMBL (unreviewed).
  4. 4
    "Identification of UAP1L1 as a critical factor for protein O-GlcNAcylation and cell proliferation in human hepatoma cells."
    Lai C.Y., Liu H., Tin K.X., Huang Y., Yeh K.H., Peng H.W., Chen H.D., He J.Y., Chiang Y.J., Liu C.S., Weng S.Y., Tao M.H., Yen J.J., Yang-Yen H.F.
    Oncogene 38:317-331(2019) [PubMed] [Europe PMC] [Abstract]
    Category: Function.
    Annotation: UAP1L1 is upregulated in a distinct subset of HCC tissues. Patients with upregulated expression of UAP1L1 have a poor prognosis. UAP1L1 overexpression promoted and UAP1L1 knockdown reduced the proliferation of human hepatoma cells both in vitro and in vivo. UAP1L1 directly interacts with OGT but is not a substrate and cannot activate it alone.UAP1L1 knockdown attenuated c-MYC O-GlcNAcylation and protein stability.
    Source: GeneRIF:91373.

    This publication is mapped to 5 other entries.

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