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Overview

Proteinsi1,024
Gene counti <p>This is the total number of unique genes found in the proteome set, algorithmically computed. For each gene, a single representative protein sequence is chosen from the proteome. Where possible, reviewed (Swiss-Prot) protein sequences are chosen as the representatives.</p> - Download one protein sequence per gene (FASTA)
Proteome IDi <p>The proteome identifier (UPID) is the unique identifier assigned to the set of proteins that constitute the <a href="http://www.uniprot.org/manual/proteomes_manual">proteome</a>. It consists of the characters ‘UP’ followed by 9 digits, is stable across releases and can therefore be used to cite a UniProt proteome.<p><a href='/help/proteome_id' target='_top'>More...</a></p>UP000001205
Taxonomy314724 - Borrelia turicatae (strain 91E135)
Strain91E135
Last modifiedSeptember 30, 2018
Genome assembly and annotationi GCA_000012085.2 from ENA/EMBL
Pan proteomei <p>A pan proteome is the full set of proteins thought to be expressed by a group of highly related organisms (e.g. multiple strains of the same bacterial species).<p><a href='/help/pan_proteomes' target='_top'>More...</a></p> This proteome is part of the Borrelia burgdorferi (strain ATCC 35210 / B31 / CIP 102532 / DSM 4680) pan proteome (fasta)

Relapsing fever (RF) is a disease caused by several spirochetes of the genus Borrelia. Relapsing fever borrelioses are characterized by recurrent febrile episodes and spirochetemia. There are 2 forms; louse-borne relapsing fever (also known as urban or epidemic RF) is caused by Borrelia recurrentis, and is transmitted by the body louse Pediculus humanus humanus. It currently known in Ethiopia. Endemic tick-borne relapsing fever (TBRF) is a zoonotic disease transmitted worldwide by softbody ticks of the genus Ornithodoros. It is caused by at least 15 distinct Borrelia species throughout the world, including Borrelia hermsii and Borrelia turicatae, carried by the ticks Ornithodoros hermsii and O.turicatae respectively (modified from PubMed 16965346). B.turicatae is pathogenic in dogs and is found in Florida, Texas and Kansas. Strain 91E135 was isolated in 1991 in Texas. Like B.hermsii, B.turicatae evades the mammalian immune system by periodically switching expression among members of two multigene families that encode immunogenic, antigenically distinct outer surface proteins (modified from 16081922).

Componentsi <p>Genomic components encoding the proteome</p>

Component nameGenome Accession(s)
Proteins
Plasmid lpu4334
Chromosome818
Plasmid lpg2927
Plasmid lpe4330
Plasmid lpt3933
Plasmid lpb3428
Plasmid cp3136
Plasmid lpf33.523
Plasmid cpd6

Publications

  1. "Reassembled and rearranged: the organization and evolution of antigen-encoding plasmids in two relapsing fever Borrelia species."
    Barbour A.G., Dai Q., Miller S.C., Porcella S.F., Schwan T.G., Lopez J.E.
    Submitted (JUL-2016) to the EMBL/GenBank/DDBJ databases
  2. "The genome sequence of Borrelia hermsii and Borrelia turicatae: comparative analysis of two agents of endemic N. America relapsing fever."
    Porcella S.F., Raffel S.J., Schrumpf M.E., Montgomery B., Smith T., Schwan T.G.
    Submitted (DEC-2004) to the EMBL/GenBank/DDBJ databases
  3. "Reassembled and rearranged: the organization and evolution of antigen-encoding plasmids in two relapsing fever Borrelia species."
    Barbour A.G., Dai Q., Miller S.C., Porcella S.F., Schwan T.G., Lopez J.E.
    Submitted (JAN-2017) to the EMBL/GenBank/DDBJ databases
  4. "Phylogenetic analysis of the spirochetes Borrelia parkeri and Borrelia turicatae and the potential for tick-borne relapsing fever in Florida."
    Schwan T.G., Raffel S.J., Schrumpf M.E., Policastro P.F., Rawlings J.A., Lane R.S., Breitschwerdt E.B., Porcella S.F.
    J. Clin. Microbiol. 2005:3851-3859(2005) [PubMed] [Europe PMC] [Abstract]
  5. "Immunologic and genetic analyses of VmpA of a neurotropic strain of Borrelia turicatae."
    Cadavid D., Pennington P.M., Kerentseva T.A., Bergstrom S., Barbour A.G.
    Infect. Immun. 1997:3352-3360(1997) [PubMed] [Europe PMC] [Abstract]
  6. "Variability of a bacterial surface protein and disease expression in a possible mouse model of systemic Lyme borreliosis."
    Cadavid D., Thomas D.D., Crawley R., Barbour A.G.
    J. Exp. Med. 1994:631-642(1994) [PubMed] [Europe PMC] [Abstract]
  7. "Extensive interplasmidic duplications change the virulence phenotype of the relapsing fever agent Borrelia turicatae."
    Pennington P.M., Cadavid D., Bunikis J., Norris S., Barbour A.G.
    Molecular Microbiology 1999:1120-1132(1999)
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