Skip Header

You are using a version of Internet Explorer that may not display all features of this website. Please upgrade to a modern browser.
Contribute Send feedback
Read comments (?) or add your own

Q71V36 (Q71V36_HUMAN) Unreviewed, UniProtKB/TrEMBL

Last modified March 19, 2014. Version 35. Feed History...

Clusters with 100%, 90%, 50% identity | Third-party data text xml rdf/xml gff fasta
to top of pageNames·Attributes·Ontologies·Sequence annotation·Sequences·References·Cross-refs·Entry infoCustomize order

Names and origin

Protein attributes

Sequence length22 AA.
Sequence statusFragment.
Protein existenceEvidence at transcript level

Ontologies

Gene Ontology (GO)
   Biological_processartery morphogenesis

Inferred from electronic annotation. Source: Ensembl

bone development

Inferred from electronic annotation. Source: Ensembl

cell migration involved in endocardial cushion formation

Inferred from electronic annotation. Source: Ensembl

cellular response to mechanical stimulus

Inferred from electronic annotation. Source: Ensembl

chronological cell aging

Inferred from electronic annotation. Source: Ensembl

extracellular matrix constituent secretion

Inferred from electronic annotation. Source: Ensembl

heart looping

Inferred from electronic annotation. Source: Ensembl

patterning of blood vessels

Inferred from electronic annotation. Source: Ensembl

positive regulation of collagen biosynthetic process

Inferred from electronic annotation. Source: Ensembl

positive regulation of gene expression

Inferred from electronic annotation. Source: Ensembl

positive regulation of protein phosphorylation

Inferred from electronic annotation. Source: Ensembl

regulation of transforming growth factor beta receptor signaling pathway

Inferred from electronic annotation. Source: Ensembl

response to corticosteroid

Inferred from electronic annotation. Source: Ensembl

response to statin

Inferred from electronic annotation. Source: Ensembl

response to transforming growth factor beta

Inferred from electronic annotation. Source: Ensembl

smooth muscle tissue development

Inferred from electronic annotation. Source: Ensembl

vasculogenesis

Inferred from electronic annotation. Source: Ensembl

venous blood vessel morphogenesis

Inferred from electronic annotation. Source: Ensembl

   Cellular_componentcell surface

Inferred from electronic annotation. Source: Ensembl

endothelial microparticle

Inferred from electronic annotation. Source: Ensembl

Complete GO annotation...

Sequence annotation (Features)

Feature keyPosition(s)LengthDescriptionGraphical viewFeature identifier

Experimental info

Non-terminal residue221 EMBL AAC32802.1

Sequences

Sequence LengthMass (Da)Tools
Q71V36 [UniParc].

Last modified July 5, 2004. Version 1.
Checksum: BE9362EF8E4F8FB4

FASTA222,230
        10         20 
MDRGTLPLAV ALLLASCSLS PT 

« Hide

References

[1]"Cloning of the promoter region of human endoglin, the target gene for hereditary hemorrhagic telangiectasia type 1."
Rius C., Smith J.D., Almendro N., Langa C., Botella L.M., Marchuk D.A., Vary C.P., Bernabeu C.
Blood 92:4677-4690(1998) [PubMed] [Europe PMC] [Abstract]
Cited for: NUCLEOTIDE SEQUENCE.
+Additional computationally mapped references.

Cross-references

Sequence databases

EMBL
GenBank
DDBJ
AF035753 Genomic DNA. Translation: AAC32802.1.
UniGeneHs.76753.

3D structure databases

ModBaseSearch...
MobiDBSearch...

Proteomic databases

PRIDEQ71V36.

Protocols and materials databases

StructuralBiologyKnowledgebaseSearch...

Gene expression databases

ArrayExpressQ71V36.
BgeeQ71V36.

Family and domain databases

ProtoNetSearch...

Other

ChiTaRSENG. human.

Entry information

Entry nameQ71V36_HUMAN
AccessionPrimary (citable) accession number: Q71V36
Entry history
Integrated into UniProtKB/TrEMBL: July 5, 2004
Last sequence update: July 5, 2004
Last modified: March 19, 2014
This is version 35 of the entry and version 1 of the sequence. [Complete history]
Entry statusUnreviewed (UniProtKB/TrEMBL)
DisclaimerAny medical or genetic information present in this entry is provided for research, educational and informational purposes only. It is not in any way intended to be used as a substitute for professional medical advice, diagnosis, treatment or care.