Q6NZ59 (Q6NZ59_HUMAN) Unreviewed, UniProtKB/TrEMBL
Last modified
November 16, 2011.
Version 43.
History...
Names·Attributes·General annotation·Ontologies·Sequences·References·Cross-refs·Entry infoCustomize order
Names·Attributes·General annotation·Ontologies·Sequences·References·Cross-refs·Entry infoCustomize orderNames and origin
| Protein names | Recommended name: ATP synthase-coupling factor 6, mitochondrial PIRNR PIRNR002455 Short name=ATPase subunit F6 PIRNR PIRNR002455 | ||
| Gene names |
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| Organism | Homo sapiens (Human) EMBL AAH66310.1 | ||
| Taxonomic identifier | 9606 [NCBI] | ||
| Taxonomic lineage | Eukaryota › Metazoa › Chordata › Craniata › Vertebrata › Euteleostomi › Mammalia › Eutheria › Euarchontoglires › Primates › Haplorrhini › Catarrhini › Hominidae › Homo |
Protein attributes
| Sequence length | 108 AA. |
| Sequence status | Complete. |
| Protein existence | Evidence at transcript level |
General annotation (Comments)
| Function | Mitochondrial membrane ATP synthase (F1F0 ATP synthase or Complex V) produces ATP from ADP in the presence of a proton gradient across the membrane which is generated by electron transport complexes of the respiratory chain By similarity. PIRNR PIRNR002455 |
| Subcellular location | Mitochondrion. Mitochondrion inner membrane By similarity PIRNR PIRNR002455. |
| Sequence similarities | Belongs to the eukaryotic ATPase subunit F6 family. PIRNR PIRNR002455 |
Ontologies
| Keywords | |
|---|---|
| Biological process | Hydrogen ion transport PIRNR PIRNR002455 Ion transport Transport |
| Cellular component | Membrane Mitochondrion Mitochondrion inner membrane PIRNR PIRNR002455 |
| Gene Ontology (GO) | |
| Biological process | ATP synthesis coupled proton transport Inferred from electronic annotation. Source: InterPro |
| Cellular component | mitochondrial proton-transporting ATP synthase complex, coupling factor F(o) Inferred from electronic annotation. Source: InterPro |
| Molecular function | hydrogen ion transmembrane transporter activity Inferred from electronic annotation. Source: InterPro |
| Complete GO annotation... | |
Sequences
References
| [1] | "The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC)." The MGC Project Team Genome Res. 14:2121-2127(2004) [PubMed: 15489334] [Abstract] Cited for: NUCLEOTIDE SEQUENCE [LARGE SCALE MRNA]. Tissue: Brain EMBL AAH66310.1. |
| + | Additional computationally mapped references. |
Cross-references
Sequence databases | |
|---|---|
| EMBL GenBank DDBJ | BC066310 mRNA. Translation: AAH66310.1. |
| IPI | IPI00002521. |
| RefSeq | NP_001003696.1. NM_001003696.1. NP_001003697.1. NM_001003697.1. NP_001003701.1. NM_001003701.1. NP_001003703.1. NM_001003703.1. NP_001676.2. NM_001685.4. |
| UniGene | Hs.246310. |
3D structure databases | |
| ProteinModelPortal | Q6NZ59. |
| SMR | Q6NZ59. Positions 33-108. |
| ModBase | Search... |
Protein-protein interaction databases | |
| STRING | Q6NZ59. |
Protocols and materials databases | |
| StructuralBiologyKnowledgebase | Search... |
Genome annotation databases | |
| GeneID | 522. |
| KEGG | hsa:522. |
Organism-specific databases | |
| CTD | 522. |
Phylogenomic databases | |
| GeneTree | ENSGT00390000008902. |
| HOVERGEN | HBG062261. |
Gene expression databases | |
| ArrayExpress | Q6NZ59. |
Family and domain databases | |
| InterPro | IPR008387. ATPase_F0-cplx_f6su_mt. IPR016349. ATPase_F0-cplx_f6su_mt_subgr. [Graphical view] |
| KO | K02131. |
| PANTHER | PTHR12441. ATPase_F0_F6_mt. 1 hit. |
| Pfam | PF05511. ATP-synt_F6. 1 hit. [Graphical view] |
| PIRSF | PIRSF002455. ATP_synthase_coupling_factor_6. 1 hit. |
| SUPFAM | SSF111357. ATPase_F0_F6_mt. 1 hit. |
| ProtoNet | Search... |
Entry information
| Entry name | Q6NZ59_HUMAN | ||||||||
| Accession | Primary (citable) accession number: Q6NZ59 | ||||||||
| Entry history |
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| Entry status | Unreviewed (UniProtKB/TrEMBL) | ||||||||
| Disclaimer | Any medical or genetic information present in this entry is provided for research, educational and informational purposes only. It is not in any way intended to be used as a substitute for professional medical advice, diagnosis, treatment or care. | ||||||||

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