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Overview

Proteome nameSalmonella agona
Proteins4,593
Proteome IDiUP000008819
StrainSL483
Taxonomy454166 - Salmonella agona (strain SL483)
Last modifiedFebruary 4, 2017
Genome assembly and annotationi GCA_000020885.1 from ENA/EMBL
Pan proteomei This proteome is part of the Salmonella typhimurium ATCC 700720 pan proteome (fasta)

Salmonella species belong to the group of Enterobactericiae. These bacteria were named after the scientist Dr. Daniel Salmon who isolated the first organism, Salmonella choleraesuis, from the intestine of a pig. The majority of the components of these bacteria are identical, and at the DNA level, they are between 95% and 99% identical. Many Salmonella enterica are involved in causing diseases of the intestine (enteric means pertaining to the intestine). The nontyphoidal Salmonella are the leading cause of bacterial food borne illness in humans, making these pathogens an immediate biomedical, public health, and biodefense concern. The presence of several pathogenicity islands (PAIs) that encode various virulence factors allows Salmonella spp. to colonize and infect host organisms. There are two important PAIs, Salmonella pathogenicity island 1 and 2 (SPI-1 and SPI-2) that encode two different type III secretion systems for the delivery of effector molecules into the host cell that result in internalization of the bacteria, which then leads to systemic spread.

Salmonella agona (strain SL483) is a major cause of human foodborne illness in the United States and around the world. It is also a major pathogen of swine and other food animals. As with many serovars of high public health importance, genetic and immunological data are lacking. S. agona has been identified recently as a serovar capable of receiving the Salmonella genomic island 1 (SGI1) multidrug resistance gene cluster by horizontal gene spread.

Componentsi

DownloadView all proteins
Component nameGenome Accession(s)
Proteins
Chromosome4541
Plasmid pSL48352

Publications

  1. "Comparative genomics of 28 Salmonella enterica isolates: evidence for CRISPR-mediated adaptive sublineage evolution."
    Fricke W.F., Mammel M.K., McDermott P.F., Tartera C., White D.G., Leclerc J.E., Ravel J., Cebula T.A.
    J. Bacteriol. 2011:3556-3568(2011) [PubMed] [Europe PMC] [Abstract]