| Cortactin promotes migration and platelet-derived growth factor-induced actin reorganization by signaling to Rho-GTPases. Lai F.P., Szczodrak M., Oelkers J.M., Ladwein M., Acconcia F., Benesch S., Auinger S., Faix J., Small J.V., Polo S. et al.
Mol. Biol. Cell 20:3209-3223(2009) · Mapped (3) |
| Cdc42 and phosphoinositide 3-kinase drive Rac-mediated actin polymerization downstream of c-Met in distinct and common pathways. Bosse T., Ehinger J., Czuchra A., Benesch S., Steffen A., Wu X., Schloen K., Niemann H.H., Scita G., Stradal T.E. et al.
Mol. Cell. Biol. 27:6615-6628(2007) · Mapped (28) |
| Enterohaemorrhagic and enteropathogenic Escherichia coli use different mechanisms for actin pedestal formation that converge on N-WASP. Lommel S., Benesch S., Rohde M., Wehland J., Rottner K.
Cell. Microbiol. 6:243-254(2004) · UniProtKB (2) |
| Phosphatidylinositol 4,5-biphosphate (PIP2)-induced vesicle movement depends on N-WASP and involves Nck, WIP, and Grb2. Benesch S., Lommel S., Steffen A., Stradal T.E.B., Scaplehorn N., Way M., Wehland J., Rottner K.
J. Biol. Chem. 277:37771-37776(2002) · UniProtKB (1) · Mapped (17) |
| Actin pedestal formation by enteropathogenic Escherichia coli and intracellular motility of Shigella flexneri are abolished in N-WASP-defective cells. Lommel S., Benesch S., Rottner K., Franz T., Wehland J., Kuehn R.
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